Barbara Tschirren

Research Interests

Evolutionary ecology, ecophysiology, life-history evolution, phenotypic plasticity, maternal effects, hormones and behaviour, host-parasite interactions, wildlife disease, immunoecology and -genetics


Education and Academic Positions

2011 - 2016 Assistant Professor (SNSF Professorship), Department of Evolutionary Biology and Environmental Studies, University of Zurich, Switzerland
2009 - 2010 SNSF Advanced Postdoctoral Fellow, Lund University, Sweden
2006 - 2008 SNSF Postdoctoral Fellow, Janggen-Pöhn Postdoctoral Fellow, Australian Research Council Postdoctoral Fellow (APD), University of New South Wales and Macquarie University, Sydney, Australia
2002 - 2005 Ph.D. in Evolutionary Ecology, University of Bern, Switzerland
2000 - 2001 Certificate in Higher Education, University of Bern, Switzerland
1999 - 2000 M.Sc. in Biology, University of Bern, Switzerland
1995 - 1999 Study of Biology, University of Bern, Switzerland

Selected Publications

  • Tschirren B., Ziegler AK., Pick JL., Okuliarová M., Zeman M. and Giraudeau M. (2016) Matrilineal inheritance of a key mediator of prenatal maternal effects. Proceedings of the Royal Society of London, Series B: Biological Sciences, 283(1838):20161676. DOI: 10.1098/rspb.2016.1676
  • Pick JL., Ebneter C., Hutter P., Tschirren B. (2016) Disentangling genetic and prenatal maternal effects on offspring size and survival. American Naturalist. DOI: 10.1086/688918
  • Tschirren B. (2015) Borrelia burgdorferi sensu lato infection pressure shapes innate immune gene evolution in natural rodent populations across Europe. Biology Letters: DOI: 10.1098/rsbl.2015.0263
  • Tschirren B., Postma E., Gustafsson L., Groothuis TGG., Doligez B. (2014) Natural selection acts in opposite ways on correlated hormonal mediators of prenatal maternal effects in a wild bird population. Ecology Letters, 17: 1310-1315. DOI: 10.1111/ele.12339
  • Tschirren B., Andersson M., Scherman K., Westerdahl H., Mittl PRE., Råberg L. (2013) Polymorphisms at the innate immune receptor TLR2 are associated with Borrelia infection in a wild rodent population. Proceedings of the Royal Society B, 280 (1759): DOI: 10.1098/rspb.2013.0364